There are approximately 15.6 million people aged 19 and under in the United Kingdom which means just 1 in every 312,000 children and teenagers have allegedly died with Covid-19 in 18 months who had other serious pre-existing conditions. Whilst just 1 in every 1.1 million children have allegedly died with Covid-19 in 20 months, who had no know pre-existing conditions.
Over the last 4 weeks in England there have been 336893 Covid cases with 6 deaths for children under 18. This gives a Case Fatality Rate of 0.001% or a survival rate of 99.999%.
Below are extracts from a number of different papers all with different reasons as to why it makes no sense whatsoever to vax the under 18’s. If vaxxing kids serves no benefit to the children or the community then whose interests other than the egotistic/psychopathic tendencies of those in power and big profits for big pharma is it been done for.
But first a quote from FDA panel member Dr. Eric Rubin in regards to vaxxing 5-11 year olds which in itself is more than enough reason to never let the jab near their arms.
“We’re never gonna learn about how safe the vaccine is until we start giving it. That’s just the way it goes.”
Now for the papers.
Lower household transmission rates of SARS-CoV-2 from children compared to adults. Source: https://www.journalofinfection.com/article/S0163-4453(21)00209-7/fulltext
An April 2021 study in the Journal of Infection (April 2021) examined household transmission rates in children and adults. The authors reported there was “no transmission from an index-person < 18 years (child) to a household contact < 18 years (child) (0/7), but 26 transmissions from adult index-cases to household contacts < 18 years (child) (26/71, SAR 0=37).”
These findings add to the stable existing evidence that children are not spreading the virus to children but rather that adults are spreading it to children. With a CFR of 0.001% infections are very little threat to life for the Under 18’s, the reasons being are numerous. First up they have a superior natural immune system, ACE2 receptors aren’t fully developed
A 2020 Yale University report indicates children and adults display very diverse and different immune system responses to SARS-CoV-2 infection which explains why they have far less illness or mortality from COVID.
Children’s immune response more effective against COVID-19. Source: https://news.yale.edu/2020/09/21/childrens-immune-response-more-effective-against-covid-19
According to the study:
“Since the earliest days of the COVID-19 outbreak, scientists have observed that children infected with the virus tend to fare much better than adults … researchers reported that levels of two immune system molecules — interleukin 17A (IL-17A), which helps mobilize immune system response during early infection, and interferon gamma (INF-g), which combats viral replication — were strongly linked to the age of the patients. The younger the patient, the higher the levels of IL-17A and INF-g, the analysis showed…these two molecules are part of the innate immune system, a more primitive, non-specific type of response activated early after infection.”
Nasal ACE2 Levels and COVID-19 in Children – Source: https://pubmed.ncbi.nlm.nih.gov/32432681/
Nasal Gene Expression of Angiotensin-Converting Enzyme 2 in Children and Adults. Source: https://jamanetwork.com/journals/jama/fullarticle/2766524
Studies by Ankit B. Patel and Dr. Supinda Bunyavanich show the virus uses the ACE 2 receptor to gain entry to the host cell, and the ACE 2 receptor has limited (less) expression and presence in the nasal epithelium in young children (potentially in upper respiratory airways).
This partly explains why children are less likely to be infected in the first place, or spread it to other children or adults, or even get severely ill. The biological molecular apparatus is simply not there in the nasopharynx of children. By bypassing this natural protection (limited nasal ACE 2 receptors in young children) and entering the shoulder deltoid, this could release vaccine, its mRNA and LNP content (e.g. PEG), and generated spike into the circulation that could then damage the endothelial lining of the blood vessels (vasculature) and cause severe allergic reactions.
Kids Don’t Need To Be Vaccinated: Proof Using CDC’S Own Numbers – Source: https://wmbriggs.com/post/37874/
The statistician William Briggs reported on the n=542 children who died (0-17 years (crude rate of 0.00007 per 100 and under 1 year old n=132, CDC data) since January 2020 with a diagnosis of COVID linked to their death. This does not indicate whether, as Johns Hopkins’ Dr. Marty Makary has been clamoring, the death was “causal or incidental.” That said, from January 2020, 1,043 children 0-17 have died of pneumonia.
Briggs reported:
“There is no good vaccine for pneumonia. But it could be avoided by keeping kids socially distanced from each other — permanently. If one death is “too many,” then you must not allow kids to be within contact of any human being who has a disease that may be passed to them, from which they may acquire pneumonia. They must also not be allowed in any car … in one year, just about 3,091 kids 0-17 died in car crashes (435 from 0-4, 847 from 5-14, and 30% of 6,031 from 15-24). Multiply these 3,000 deaths in cars by about 1.75, since the COVID deaths are over a 21-month period. That makes about 5,250 kids dying in car crashes in the same period — 10 times as many as Covid.”
Briggs concluded: “there exists no justification based on any available evidence for mandatory vaccines for kids.”
Distinct antibody responses to SARS-CoV-2 in children and adults across the COVID-19 clinical spectrum. Source: https://www.nature.com/articles/s41590-020-00826-9?campaign_id=154&emc=edit_cb_20201106&instance_id=23895&nl=coronavirus-briefing®i_id=61609026&segment_id=43815&te=1&user_id=73aefc51739a420f3d188e5
Weisberg and Farber et al. suggest (and building on research work by Kumar and Faber) that the reason children can more easily neutralize the virus is that their T cells are relatively naïve. They argue that since children’s T cells are mostly untrained, they can thus immunologically respond (optimally differentiate) more rapidly and nimbly to novel viruses such as SARS-CoV-2 for an effective robust response.
Pre-activated antiviral innate immunity in the upper airways controls early SARS-CoV-2 infection in children. Source: https://www.nature.com/articles/s41587-021-01037-9
Research published in August 2021 by J. Loske deepens our understanding of this natural type biological/molecular protection even further by showing that “pre-activated (primed) antiviral innate immunity in the upper airways of children work to control early SARS-CoV-2 infection … the airway immune cells in children are primed for virus sensing…resulting in a stronger early innate antiviral response to SARS-CoV-2 infection than in adults.”
Shared B cell memory to coronaviruses and other pathogens varies in human age groups and tissues. Source: https://www.science.org/doi/10.1126/science.abf6648
When one is vaccinated or becomes infected naturally, this drives the formation, tissue distribution and clonal evolution of B cells, which is key to encoding humoral immune memory.
Research published in May 2021 showed that blood examined from children retrieved prior to COVID-19 pandemic have memory B cells that can bind to SARS-CoV-2, suggestive of the potent role of early childhood exposure to common cold coronaviruses (coronaviruses). This is supported by Mateus et al. who reported on T cell memory to prior coronaviruses that cause the common cold (cross-reactivity/cross-protection).